If you can’t understand that non binary is just an extension of the gender binary that does nothing to abolish gender but actually re-inforces it idk how to help you

From my take as a non-binary person and gender non confirming person, this is the modern notion and acknowledgement that a gender binary has existed and will continue to exist for some people while giving those who do not want to live in the gender binary an opportunity to live outside of it. There is plenty of historical and cultural argument against the gender binary with identities such as Two Spirit (Native American), Hijras (South Asia), and Il Femminielo (Italy). Many in LGBTQ+ studies argue that the gender binary is therefore colonialist due to the number of gender variant identities outside of Western Europe. Even the identity of intersex defeats the notion that there is some kind of biological identity of male and female.

I thought these talking points had all been so heavily debunked that they weren’t even worth talking anymore but apparently not lmao. People are really still saying this shit.

Intersex is not an identity, what the fuck. It’s an umbrella term for multiple medical conditions you are born with, not some mysterious third sex. Not to mention that people with intersex disorders have asked approximately 10000000 times not to be used as misappropriated props in your stupid gender rationalizations.

There’s a person on Twitter who (I’m not sure if they’re intersex) basically attests to this and has written about the issue:

@inferior-mirage and @radfemflareon are two of the best sources for intersex information on here, IMO. They always have really great articles and super detailed explanations, and I’ve learned a lot of stuff I didn’t know from their blogs.

While she’s not currently active I also recommend taking a read through the blog of @star-mut who unlike Mirage and I has the experience of living with a sex reversal disorder, being genetically male but an external female phenotype. She speaks more in the way of how gender theory ushered in by John Money affects intersex lives than of how intersex disorders work, like I focus on. She’s got far more knowledge in the way of intersex rights politics than I do as someone with only a mild variant and with only two decades on this earth. She’s been here for the entirety of the birth and rise of modern queer theory and its engulfment of intersex activism and offers great perspective on how the cogwheels are turning.


I didn’t come out until I was 20 and had my first kiss at 21 and I am now extremely happily married and comfortably a Huge Dyke

and I know women who didn’t start to come to terms with their lesbianism until they were in their 30s who now have awesome girlfriends/wives

so, teen lesbians, don’t worry! you’ve got so much time to date and smooch and enjoy pussy and find love and all that other good romance and sex stuff! there is no deadline 💜







i have known multiple men irl, including my own stepfather, who like to claim that being the husband of a woman who is pregnant/giving birth is just as difficult as being a woman who is pregnant/giving birth

every time i think about this i want to stop and shake them and yell “you do realise that women die in childbirth still, to this day???”

but i know that the real answer here is that these kinds of men can’t bear not being the center of attention for even a minute, and so they have to make this about themselves and how much they “suffer”

Yeah but like, he has to do things for her and she can’t do everything for him! That’s hard! ): 

feeling obligated to support the mother of your child is just as bad as preeclampsia guys 😦

“Woe is me, me having to wait on my lazy pregnant wife is just as bad as pushing a baby the size of a toaster down a tube the width of a garden hose!!! Pity meee 😭”

That’s literally in the bible though. The ultimate punishment for disobeying god for women: painful menstruation and excruciating dangerous birth. The ultimate punishment for disobeying god for men: having to listen to women complain about it. Sure, seems about equal to me.🙄

My friend’s husband went through a “sympathetic pregnancy” when she had their first child and everyone treated him like the most wonderful loving partner because he whined about his nips being sore. His mother was in charge of the baby shower and my friend received two of everything (heating pad for post-pregnancy cramps, bag balm for breastfeeding wounds, etc.) so her husband could have one too. Then when she went into labor, everyone’s more concerned about HIM because “his belly really hurts and that must mean something important, unlike this woman who is currently expelling a watermelon!” They made a huge fuss about making him comfy in the delivery room before they even started her IV or anything. It was the most ridiculous thing I’d ever seen.

in my honest opinion, ‘sympathetic pregnancy" is nothing more than a symptom of a narcissistic husband who can’t bear being out of the spotlight for even two seconds.

This honestly sounds like he was garnering narcissistic supply while secretly indulging in some sort of fetish. God I feel so sorry for his wife.





“Why should I have to prove I’m “trans enough” to get hrt,“

I dunno probably for the same reason you have to prove you’re depressed before you get anti-depressants or have to prove you have insuline deficiency before you get insuline shots.

it took more testing for me to get on ADD meds than it did hormones, one of which is out of my system in 24 hours and the other fucked up my body and left me with permanent changes that can only be somewhat reversed by medical interventions.

Can I be a little nosey and ask for details? Did you go through informed consent? Were you diagnosed with dysphoria? Are you detransitioning?

Sure. I was diagnosed with dysphoria by about three or four different therapists from the time I was about 15. When I finally decided to medically transition, I did go through informed consent, because I had moved somewhere that was too far from any official gender clinics by that time. While I absolutely could have gone to one of these places and gotten the approvals necessary, Planned Parenthood only required that I sign a document saying I was seeing a therapist (not even a gender specialist, just a therapist of any kind, by the way), sign a for form saying I understood the risks, take a quick blood test, and within two weeks of my official phone call to them, I had my first shot. They would have been able to give it to me the same day I signed the documents if I’d done the blood tests through a different doctor ahead of time and just given them the results. It was just as easy to get two doctors to sign off my on my top-surgery, which also kind of disturbs me in hindsight.

Unlike my need for ADD medication which, despite being told I fit most of the criteria for it, took several doctors to get taken seriously, multiple psychiatrists to try and schedule the test only to be told they didn’t think I needed it because I was getting decent enough grades in school, and then several months of waiting once a psychiatrist DID take me seriously in order to get the test and finally get on the medication. I then had to sign a bunch of forms and wait an extra session afterward to make sure that I fully understood the risks of stimulants before my psychiatrist was finally allowed to put in the prescription for my medication.

I am detransitioning now; my transition ultimately didn’t help my dysphoria much and I was having pretty bad medical side effects from long term testosterone use, so I didn’t want to continue to harm my body when it wasn’t helping. Kind of like how my depression isn’t helped by any known depression medications well enough for me to suffer through the side effects I get from them, so I choose to find alternate means to manage both.

If you’ve got more questions, I’m happy to answer them. I agree with your original post btw in case it comes across like I don’t somehow – I think it’s messed up that you already have to not prove your “trans enough” to transition, given that even people who do go through all those hoops can still have regrets (albeit in much smaller percentages; I admit I’m an anomaly), and I can only imagine how much higher those regrets are in people who go on HRT and were never properly diagnosed at all.






And they shared the whole story. 

Reblog everytime..

I only wish his name/face was attached to this post too!


I read the full story and this wasn’t the 1st or even 2nd time that he had drugged her with ecstacy, she realized in retrospect. He also created pornographic content without women’s consent and he obsessively pursued this victim after she got a new boyfriend.

This guy felt so impervious based on his previous behavior that he slipped a drug into her drink in broad daylight and would have 100% gotten away with it if these bystanders hadn’t intervened.









doctors: GOOD NEWS EVERYONE we have found a treatment for diabetes it’s called insulin! people won’t die from this illness anymore if we just give them insulin isn’t this great news?

united states of america: how about we make it so poor people have either limited to zero access to this insulin?

doctors: but don’t we have enough resources to provide for everyone in need of such a medication?

the united states of america: yes!

doctors: isn’t that genocide

united states of america: YES

united states of america fist pumping and chanting ‘U’ ‘S’ ‘A’:

capitalism is genocide

i usually don’t add to posts i reblog but as a type 1 diabetic this makes me so fucking angry.

i have an insulin pump. each time i change the port (basically what connects it to my body, similar to an iv) i use about a third of a bottle refilling the cartridge. i change it every three days, so one single, 10ml bottle of insulin lasts me around nine days.

i had to look this up because my parents don’t like to tell me how much my supplies cost. (for reasons like this. i feel guilty for my t1d. i shouldn’t have to feel guilty about an autoimmune disorder i was born with.)

you know how much it is for that one, single 10ml bottle of insulin, for those without any insurance?


for nine days.

$328 for a nine days supply of the medicine that i literally need to survive. the medicine that once i become an adult and have to take care of myself, i will have to pay for. the medicine that unless, by some miracle, they find a cure, i will need to take for the rest of my life. $328 for nine days of my life.

Near and dear to my non functional pancreas 

Fam, I am also type 1 diabetic. I also use a pump. I am preparing to go do fieldwork in Europe for nine months and insulin is perishable. I use a brand that’s available in Europe. The same vial that costs $300+ in the US without insurance costs €22 ($25.60) in one country and $10-15 in the other. Not with insurance! Cash. Roll up to the pharmacy and explain that you have diabetes cash price.

It doesn’t have to be like this. The US needs to start negotiating better pharmaceutical prices like every other country. Get some socialism up in this bitch.


>_> Pretty much.


Hi! So on one of your posts it said: “CAH is also tied to high rates of same sex attraction in girls and the more masculinizing the disorder the more exclusive that attraction becomes, as well as causing aggression in forms like “boys’ play” in childhood. “ Could you tell us more about that? Or give some links to read up on that? Because I never knew about the Ashkenazi link or anything!

For sure I’ll explain some! I found a few different studies on CAH and female same sex attraction here: (1) (2) (3)

A summarizing quote from Source 1, from 2008:

“…Rates of bisexual and homosexual orientation were increased above controls not only in women with classical CAH, but also in NC women, and correlated with the degree of prenatal androgenization.”

“Bisexual/homosexual orientation was (modestly) correlated with global measures of masculinization of non-sexual behavior and predicted independently by the degree of both prenatal androgenization and masculinization of childhood behavior. We conclude that the findings support a sexual-differentiation perspective involving prenatal androgens on the development of sexual orientation.”

From Source 2, circa 1992:

“These results corroborate earlier reports on both delays in reaching psychosexual milestones and increased rates of bisexual/homosexual fantasies and experiences in CAH women.”

And from Source 3, which doesn’t focus entirely on CAH but quotes from study (J Sex Res 2004; 41: 75-81) from 2004:

“Congenital adrenal hyperplasia
(CAH) is often (90%) associated with increased DHEA. It was recently reported
that “among women with CAH, we found that recalled male-typical play in
childhood correlated with reduced satisfaction with the female gender and
reduced heterosexual interest in adulthood. Although prospective studies are
needed, these results suggest that those girls with CAH who show the greatest
alterations in childhood play behavior may be the most likely to develop a
bisexual or homosexual orientation as adults and to be dissatisfied with the
female sex of assignment.” (J Sex Res 2004; 41: 78-81). I suggest this
fits my hypothesis, that is, that increased DHEA in utero increases
“male” orientation, growth and development of the pertinent part of
the brain, in these girls. These girls are like boys in early play and later
sexual orientation. The testosterone of puberty in these girls will simply
intensify their orientation. Testosterone levels, on average, do not differ
between heterosexual and homosexual women (Horm Behav 1987; 21: 347-57). The
difference occurs in utero. (A male with extra DHEA in utero would simply have
more “male” orientation.) No differences in CAH male childhood play
or sexual orientation are found (J Sex Res 2004; 41: 75-81).”

This last link is also a very good read on some of the biological development behind sexuality.

As for frequency among Ashkenazi Jewish people, here is the source for the 1 in 27 affected and 1 in 3 carrier frequencies.

Carrier Frequency: 1 in 3 in the Ashkenazi Jewish population
1 in 61 worldwide
Disease Frequency: 1 in 27 in the Ashkenazi Jewish population
1 in 100 in non-Ashkenazi Jews.
1 in 15,000 worldwide

CAH is an autosomal recessive disorder that effects certain ethnicities more than others just like black people are more likely to have sickle cell anemia, another genetic disorder. The most common population for CAH is the Ashkenazi, but it has decent frequency in Hispanic and Italian people as well.

A massive study on 1,500 families with members having CAH and their mutation genotypes and disorder severity can be found here. This source also gives us good information on which mutations are more commonly found in which ethnicities.

“…There was a high frequency of homozygous I2G [Intron 2 Splice] and V281L mutations in Middle Eastern and Ashkenazi Jewish populations, respectively.”

“One critical distinction between our data and three previous studies is the enrichment of the exon 7 V281L mutation noted in our cohort. Only one study presents a similar frequency for this mutation (21). This enrichment is likely due to the large number of Ashkenazi Jews within our population, who we find have a significantly higher frequency of this mutation (Table 2).”

Table 2 illustrates the ethnic diversity of CAH genotypes. We note that certain genotypes are more frequent in specific ethnic groups. For example, the I2G/I2G genotype is more prevalent in the Middle Eastern population. As noted before, the exon 7 V281L mutation associated with NC CAH is very frequent in the Ashkenazi Jewish population. We also find that CAH is rare in Asian, African American, and East Indian populations in this cohort.”

The rarest form of CAH, lipoid CAH, affects almost always Koreans with a few Japanese and Middle Eastern patients. Lipoid CAH has only 97 confirmed cases (source).

“To date, at least 97 patients with LCAH and more than 35 different mutations have been reported in the STARD1 gene (Bhangoo et al., 2005, 2006; Papadimitriou et al., 2006; Abdulhadi-Atwan et al., 2007).”

“More than 70% of Japanese alleles and all Korean alleles carry the Q258X mutation; a second genetic cluster is found among Palestinian Arabs, most of whom carry the R182L mutation. Many publications underline that some correlations exist between the severity of STARD1 mutation and the age of onset of a clinical salt-wasting crisis, hypoglycemia and gonadal function (Bose et al., 1996; Miller, 1997; Bhangoo et al., 2005, 2006). Patients with the mutation Q258X or R182L are generally symptomatic by the age of 1 day to some weeks. Three unrelated patients of Swiss ancestry, L260P mutant, had their onset of symptoms at the age 2.5–5.5 months (Flück et al., 2005). Furthermore, as reported by Chen et al. (2005) patients from eastern Saudi Arabia who carry the R182H mutation, very close to the Palestinian mutation R182L, had a milder disease starting at the age of 1–14 months, and 4 out of 7 of them had the onset of their symptoms from 7–14 months of age; functional study in this group showed a complete loss of STARD1 activity. One patient from Qatar, with R182H mutation, had the onset of clinical symptoms and laboratory evidence of salt loss at the age of 3 weeks (Achermann et al., 2001); the in vitro STARD1 activity of this patient was not determined.”

And on the topic of mutations being very specific to ethnicities and severities, I’d like to mention that while I am homozygous for V281L which genotypically designates me as nonclassical, there are some instances of the same mutation manifesting more severely and causing adrenal crises though these are rare. This may be the explanation for my medical history, to clarify for anyone who may come across my blog and see that I’m a NCAH patient who’s had crises. The quote for that is found in the 1,500 family study on genotype-phenotype correlation:

“However, although 98% of cases are NC, a smaller number of patients do present with classical CAH (Fig. 3; Table S4), suggesting that genotype–phenotype or structure–phenotype correlations in CAH are usually not perfect. Sanger sequencing (28) of all nine cases of classical CAH with the V281L mutation showed no other mutation from the proximal promoter to the proximal 3′ UTR region in the CYP21A2 gene to account for the phenotypic variation. We are unclear of the mechanism through which a ∼50% reduction in 21-hydroxylase activity results in classical CAH.”